Jörg Striessnig

Corresponding Member of the Division of Mathematics and the Natural Sciences in Austria since 2004
- Institut für Pharmazie, Universität Innsbruck
Contact:
joerg.striessnig(at)uibk.ac.at
Research Areas:
- Medical-Theoretical Sciences, Pharmacy
- Pharmacology
- Neuropharmacology
- Molecular pathology
Profile:
Selected Memberships:
- Deutsche Akademie der Naturforscher Leopoldina - Nationale Akademie der Wissenschaften
- Academy of Europe - Academia Europaea
Selected Prizes:
- International Albrecht Fleckenstein Award
- Tiroler Landespreis für Wissenschaft
- Max-Kade Fellowship (Austrian Academy of Sciences)
- ISI Highly Cited Researcher
Selected Publications:
- Azizan, E. A.; Poulsen, H.; Tuluc, P.; Zhou, J.; Clausen, M. V. et al. [..] (online: 2013 Somatic mutations in ATP1A1 and CACNA1D underlie a common subtype of adrenal hypertension. Nature Genetics, Bd. 45, S. 1055–1060.
- Baig, S. M.; Koschak, A.; Lieb, A.; Gebhart, M.; Dafinger, C. et al. [..] (2011) Loss of Cav1.3 (CACNA1D) function in a human channelopathy with bradycardia and congenital deafness. Nature Neuroscience, Bd. 14, S. 77-84.
- Singh, A.; Hamedinger, D.; Hoda, J. C.; Gebhart, M.; Koschak, A. et al. [..] (2006) C-terminal modulator controls Ca2+ -dependent gating of Cav1.4 L-type Ca2+ channels. Nature Neuroscience, Bd. 9, S. 1108-1116.
- Platzer, J.; Engel, J.; Schrott-Fischer, A.; Stephan, K.; Bova, S. et al. [..] (2000) Congenital deafness and sinoatrial node dysfunction in mice lacking class D L-type Ca2+ channels. Cell, Bd. 102, S. 89-97.
- Siller A, Hofer NT, Tomagra G, Burkert N, Hess S, et al. [.] (2022) β2-subunit alternative splicing stabilizes Cav2.3 Ca2+ channel activity during continuous midbrain dopamine neuron-like activity. Elife, Bd. 11, S. e67464.